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Cet abstract a été assigné à session BGS : Belgian Glaucoma Society
TitreGlaucoma masquerades
Abstract Nr.101
ButIf the optic disc looks glaucomatous but the IOP is normal, one must look carefully at the neuroretinal rim. If pallor is present, the pseudoglaucomas
-compressive or previous ischemic optic neuropathy, trauma, demyelination, toxin, syphilis, dominant optic neuropathy, Leber optic neuropathy or radic optic neuropathy - must be considered.
If the increased cup/disc ratio is no accompagnied by pallor of the neuroretinal rim, one can look for undetected diurnal variation in IOP or previously elevated IOP from undiagnosed secondary glaucoma or burned-out chronic open-angle glaucoma. Other causes of intermittent elevations of IOP must be considered : total inversion (head standing), subacute angle closure attacks, glaucomatocyclitic crisis, topical steroïd therapy or spontaneously resolved pigmentary glaucoma. If still no diagnosis can be made, the ophthalmologist must rule out congenital optic disc disorders that may mimic glaucomatous cupping : coloboma, optic pits, megalopapilla, tilted discs, disc drusen, myopic optic disc and morning glory disc. If these are not present and syphilis has been ruled out with a negative FTA-ABS test, the diagnosis of low-tension glaucoma (LTG) can be made.
Neuroimaging should therefore be performed in patients who do not fit the profile of low tension glaucoma.
Auteur 1
NomCOLLIGNON
InitialesN
InstitutULg
VilleLiège
Auteur 2
NomKESTELYN
InitialesPh
InstitutUGent
VilleGent
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